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Spinal Cord

Tethered cord refers to an abnormal attachment of the spinal cord at its distal end (27). Under normal circumstances, the conus medullaris ascends from its distal position to the L1 to L2 vertebral level dur­ing the first year of life (37).

Focal abnormalities— including thickened and shortened filum terminale, supernumerary fibrous bands, persistent membrane reunions, dural sinus, diastematomyelia, entrapment by lumbosacral tumors, and adhesions in the scar tis­sue of the repaired myelomeningocele—interfere with this process (27). All children born with spina bifida have a low-lying cord on magnetic resonance imaging, and approximately one-third develop neurologic, uro­logic, or orthopedic complications or symptoms (38) (Fig. 9.2).

Tethering of the spinal cord is the second most common cause of neurologic decline in a child with myelomeningocele (38). The most common clin­ical signs or symptoms of a tethered cord include spasticity in the lower extremities, decline in lower extremity strength, and worsening scoliosis. Other signs and symptoms that strongly suggest tether­ing of the spinal cord include back pain, changes in urologic function, changes in gait, and develop­ment of lower extremity contractures. In patients who are suspected of having a symptomatic teth­ered cord, the function of their shunt needs to be evaluated prior to proceeding forward with surgical management (39).

The reported functional outcome of surgical man­agement of a tethered cord is variable. One study reported improvements in gait in almost 80% of patients following untethering, whereas other stud­ies report improvement in as few as 7% (40). (Note: All cords tether to some extent following repair.) Less than 20% of children with a tethered cord experience back pain. However, this is the symptom most likely to improve with surgery (30,41).

Diastematomyelia is a postneurulation defect that results in a sagittal cleavage of the spinal chord, most commonly affecting the lumbar and thoracolumbar levels of the spinal cord.

It is more common in females (42,43). Diastematomyelia may have both neurologic and orthopedic presentations. Orthopedic symptoms include scoliosis, Sprengel's deformity (especially when associated with Klippel-Feil sequence), hip sub­luxation, and lower extremity limb-length discrepan­cies (43,44).

Associated Central Nervous System Malformations

Spinal cord

Tethering

Distal focal abnormalities

Thick, short filum terminale

Supernumerary fibrous bands

Lumbosacral tumors (lipoma, fibrolipoma, fibroma dermoid, epidermoid cyst, teratoma)

Bony vertebral ridge

Diastematomyelia, diplomyelia, split cord

Cerebellum

Arnold-Chiari type II malformation

Elongated vermis, inferior displacement

Herniation into cervical spinal canal Abnormal nuclear structures

Dysplasia, heterotopia, heterotaxia

Ventricular system

Hydrocephalus

Aqueductal stenosis, forking, atresias

Brainstem

Arnold type II malformation

Kinking, inferior displacement of medulla

Herniation into cervical spinal canal Abnormalities of nuclear structures

Dysgenesis, hypoplasia, aplasia, defective myelination Hemorrhage, ischemic necrosis Syringobulbia

Forebrain

Polymicrogyria

Abnormal nuclear structures

Heterotopia (subependymal nodules)

Heterotaxia

Prominent massa intermedia

Thalamic fusion

Agenesis of olfactory bulbs and tracts Attenuation/dysgenesis of corpus callosum

9.1

Figure 9.2 T2-weighted magnetic resonance image of tethered cord. There is tethering of the spinal cord with conus seen down to the L5 vertebral level, heterogeneous signal intensity characteristics, and areas of fibrofatty tissue.

A

Neurologic symptoms include gait abnormalities, asymmetric motor and sensory deficits of the lower extremities, and neurogenic bladder and bowel (45).

Symptoms of diastematomyelia may present in child­hood or, less commonly, in adulthood (46).

It is not uncommon for individuals to develop syringomyelia—a tubular cavitation in the spinal cord parenchyma extending more than two spi­nal segments (47). Syringomyelia is present in up to 40% of individuals with myelomeningocele (48). The syrinx may be located anywhere along the spi­nal cord, medulla, or pons, but is most common in the cervical region (23,24,49). Magnetic resonance imaging (MRI) is used to detect syringomyelia (50) (Fig. 9.3).

Often, a syrinx is of little clinical significance; however, if a patient develops decreasing function above the level of their lesion, syringomyelia must be considered in the differential diagnosis. Although shunt malfunction and cord tethering are more com­mon complications, symptomatic hydromyelia may explain a slower-than-expected progression through gross motor and fine motor developmental milestones or a decrease in strength/function. Early progression of scoliosis above the initial neurologic level may be the earliest sign of a syrinx. A shunt malfunction

B

Figure 9.3 T2-weighted magnetic resonance image showing sagittal (A) and axial (B) views. There is a large syrinx present, beginning at the mid portion of C6 and extending to L4.

may contribute to a symptomatic syrinx, and shunt function should be evaluated. Placement of a syrin- gopleural shunt may be necessary to decompress the syrinx.

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Source: Alexander M.A., Matthews D.J.. Pediatric Rehabilitation: Principles and Practice. 4 th. åd. — New York: Demos Medical Publishing,2010. — 540 ð.. 2010
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