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Myocarditis or Acute Coronary Syndrome?

It is well known that acute myocarditis can clinically masquerade as acute MI. In the setting of acute chest pain with concomitant ST-segment elevation on at least two con­tiguous ECG leads, guidelines for therapeu­tic management of ST-segment elevation MI should be applied.

We suggest that invasive coronary angiography should be the pre­ferred method when rapidly available in order to avoid inappropriate thrombolytic therapy. CMR should play an important role when coronary angiography rules out signif­icant coronary stenosis in these cases and has the potential to confirm the diagnosis of myocarditis. On the other hand, in the set­ting of acute chest pain without ST-segment elevation, CMR if rapidly available may become the first line imaging study, espe­cially in those patients with low risk profile and/or recent history of flu.

Myocardial edema may be depicted on T2-weighted black-blood spin echo CMR during the acute phase of MI. Although myocardial distribution of edema is theoret­ically different, it is often difficult to distin­guish between myocarditis and acute MI based on T2-weighted CMR images. First- pass myocardial perfusion imaging may help distinguish the two diseases as there is no early subendocardial defect in myocardi­tis, whereas it is very common in acute MI despite prompt reopening of the infarct- related-artery (60% of cases). As previously described, patterns of delayed hyperen­hancement are very distinct and the most useful to discriminate between acute MI and acute myocarditis (Table 1).

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Source: Barbaro Giuseppe, Boccara Franc (eds.). Cardiovascular Disease in AIDS. 2nd edition. — Springer,2009. — 169 p.. 2009
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  3. Barbaro Giuseppe, Boccara Franc (eds.). Cardiovascular Disease in AIDS. 2nd edition. — Springer,2009. — 169 p., 2009