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Pancreatic cystic lesions

Pathogenesis and description

Cystic lesions are uncommon in veterinary medicine. Although rare, cystic pancreatic neoplasia should be included as a differential diagnosis of cystic intra-abdominal and pancreatic masses (Yoshimura et al., 2013; Torner et al., 2020).

The majority of cystic structures are pseudocysts (VanEnkevort et al., 1999; Charles, 2007). Pseudocysts can be defined as intra- or peripancreatic, fluctuant pockets of pancreatic enzyme secretions, necrotic debris formed by destruction and necrosis of pancreatic tissue, usually caused by pancreatitis or pancreatic trauma. Pseudocysts lack the epithelial lining characteristic of true cysts (Branter et al., 2010). Instead, the wall of a pseudocyst is composed of granulation tissue that can mature into fibrous scar tissue. The lumen contains cellular debris and pancreatic enzymes (Meuten, 2002; Head, 2003; Charles, 2007; Zachary et al., 2013). Pseudocysts are filled with often turbid, sometimes blood-tinged fluid. When a cystic lesion is visualized by ultrasound, the steps necessary to confirm a pseudocyst include a high cystic fluid amylase and/or lipase activity, the exclusion of an abscess by FNA, and histology of both the pancreas and the wall of the lesion. However, no information is available on pseudocystic fluid canine pancreatic lipase (cPL) and feline pancreatic lipase (fPL) concentrations.

Additional non-neoplastic cavitary lesions of the pancreas include abscesses, congenital cysts, and retention cysts (Table 8.1) (Coleman et al., 2005; Anderson et al., 2008; Branter & Viviano, 2010). True cysts, like pseudocysts, are variably sized, fluid-filled lesions demarcated by a wall and located in or close to the pancreatic parenchyma (Coleman et al., 2005; Charles, 2007; Branter & Viviano, 2010; Zachary & McGavin, 2013). True cysts can be unilocular or multilocular thin-walled sacs that range from millimeters to several centimeters in diameter and often contain a clear and translucent serous fluid (Bergin et al., 2002; Meuten, 2002; Head, 2003; Coleman et al., 2005).

Histologically, pancreatic cysts are lined by a single layer of low cuboidal to flattened well-differentiated ductal epithelial cells (Head, 2003; Coleman et al., 2005; Charles, 2007). True cysts can be the result of congenital ductular malformations (cystic dilation of pancreatic ducts often in association with polycystic kidney and/or liver diseases) (Meuten, 2002; Head, 2003; Charles, 2007; Zachary & McGavin, 2013) or can be acquired due to obstruction of the duct system and retention of pancreatic secretion (Meuten, 2002; Head, 2003). True congenital pancreatic cysts are extremely rare. Few cases have been reported in cats, and only one in a dog (Healy et al., 2022) They can be defined as a fluid-filled cyst lined with cuboidal-to-columnar epithelium and a cystic amylase concentration of juvenile acinar atrophy; primary (starvation…) or secondary atrophy (inflammation, neoplasia…); multifocal degeneration/necrosis (viruses, toxics, ductal obstruction...); lipofuscinosis; vacuolation (lysosomal storage diseases); acquired retention cyst (due to obstruction of ductular lumen) Hydropic degeneration; amyloidosis; necrosis (selective or by extension of acute pancreatic necrosis/pancreatitis); sclerosis Inflammatory lesions Atrophic lymphocytic pancreatitis (first stage of canine juvenile acinar atrophy); acute lesions: acute suppurative pancreatitis/acute pancreatic necrosis/acute necrotizing pancreatitis; chronic lesions: chronic nonsuppurative pancreatitis/pancreatic fibrosis; sequelae: pseudocyst/abscess/phlegmon; ductular inflammation (parasitism, lithiasis) Hyperplastic lesions Ductal hyperplasia; nodular acinar hyperplasia Islet hyperplasia; nesidioblastosis (combined ductular and islet cells hyperplasia) Neoplastic lesions Epithelial: exocrine adenoma/adenocarcinoma; non-epithelial: fibroma/sarcoma, hemangiosarcoma, liposarcoma, nerve sheath tumors, lymphoma; secondary tumors (metastasis or direct extension) Islet cell adenoma/carcinoma (insulinoma, gastrinoma, glucagonoma, pancreatic polypetide-secreting tumor) (1–5) 1. Meuten DJ.
Tumors in domestic animals. Ames, IA: Iowa State Press; 2002. 2. Zachary JF, McGavin D. Pathologic Basis of Veterinary Disease: Elsevier Health Sciences; 2013. 3. Head KW. Histological classification of tumors of the alimentary system of domestic animals: Published by the Armed Forces Institute of Pathology in cooperation with the American Registry of Pathology and the World Health Organization Collaborating Center for Worldwide Reference on Comparative Oncology; 2003. 4. Charles JA. Pancreas. In: M.; GM, editor. Jubb, Kennedy, and Palmer's Pathology of Domestic Animals, Fifth edition. Philadelphia: Elsevier Saunders; 2007. 5. Ramirez et al, JCP, 2013 “Intrapancreatic ectopic splenic tissue in dogs and cats”.

Ultrasound

True cysts, pseudocysts, abscesses, and cystic neoplasia may be difficult to differentiate on the sole basis of sonographic appearance (Larson, 2016; Griffin, 2020). Round to oval shape, thin and sharply demarcated wall, anechoic content, and strong distal acoustic enhancement are the usual and classical descriptions of cystic sonographic images. However, in the particular case of pancreatic pseudocysts, irregular margins, thick wall, and echoic content with slight acoustic enhancement (fluid rich in enzymes and debris) are possible findings. Obtaining a sample of cyst content under ultrasound guidance can help rule out abscess or cystic neoplasia. Aspiration and drainage of pseudocysts are reported to be safe and should be considered for the evaluation of cystic pancreatic lesions (VanEnkevort et al., 1999).

Cytology

As for any cyst or pseudocyst from any organ or structure, cytology is often of low cellularity, and a clear to slightly hazy fluid is obtained, suggestive of a modified transudate. In contrast with abscesses, cystic samples are composed of an abundant amorphous background and few inflammatory cells, such as few non-degenerated neutrophils and reactive macrophages (VanEnkevort et al., 1999; Bjorneby & Kari, 2002; Borjesson, 2014). One description of a congenital cyst in a dog reports a similar composition with a proteinaceous fluid associated with necrotic cellular material and mild non-septic neutrophilic inflammation with moderate to marked accumulation of uniformly blue-black pigment (Healy et al., 2022). Cytology should be considered as an exclusionary diagnostic tool for abscess.

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Source: Barger A.M., MacNeill A.L. (Eds.). Small Animal Cytologic Diagnosis: Canine and Feline Disease. CRC Press,2024. — 536 p.. 2024
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